For decades, we've been told that chronic disease is a result of genetics, poor diet, and lack of exercise. While these factors play a role, a growing body of research points to a more insidious culprit hiding in plain sight.

The Invisible Threat
Recent studies published in major medical journals have found a disturbing correlation between the presence of microplastics in the human body and markers of chronic inflammation. These tiny particles, smaller than a grain of sand, have been detected in human blood, lungs, and even the placenta.
"The chemicals contained in microplastics have been shown to accumulate in bodily tissues" says Dr. Eve Glazier, an Internal Medicine Doctor at UCLA. "This can lead to bioactive effects, including causing inflammation, changing hormone interactions, altering immune response, disrupting the gut microbiome and causing physical damage."
Key Findings:
- 93% of Americans have detectable levels of BPA and other plastic-related chemicals in their bodies.
- Microplastics have been shown to disrupt the endocrine system, potentially leading to hormonal imbalances.
- Chronic inflammation caused by foreign particles is a known precursor to autoimmune diseases.
Connecting the Dots
The rise in chronic diseases—including diabetes, obesity, and autoimmune disorders—parallels the exponential growth of plastic production over the last 50 years. While correlation does not equal causation, the biological mechanisms are becoming clearer.
When microplastics enter the gut, they can compromise the intestinal barrier (leaky gut), allowing toxins to enter the bloodstream. This triggers an immune response that, over time, can lead to systemic inflammation.
What Can Be Done?
While completely avoiding microplastics is nearly impossible in the modern world, experts suggest focusing on reduction and elimination.
- Filter your drinking water with high-quality filtration systems.
- Avoid heating food in plastic containers.
- Choose natural fibers for clothing and bedding.
- Support your body's natural detoxification pathways.